Neuronal migration (8 items)

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1. PAR-aPKC complex disruption causes a ventral mismigration in which FBMNs escape the hindbrain ventrally © Grant and Moens; licensee BioMed Central Ltd.

2. In the absence of Laminin, FBMNs fail to speed up and do not reorient centrosomes © Grant and Moens; licensee BioMed Central Ltd.

3. Removal of aPKC or Pard6gb results in defects of apical midline formation and maintenance © Grant and Moens; licensee BioMed Central Ltd.

4. FBMNs exit the hindbrain through holes in the ventral Laminin-containing basement membrane that can be phenocopied by Laminin knockdown © Grant and Moens; licensee BioMed Central Ltd.

5. aPKC functions cell non-autonomously to prevent ventral mismigration © Grant and Moens; licensee BioMed Central Ltd.

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